|
|
Joined: May 2008
Posts: 4,707
Master Elite Member
|
Is Central Hyperacusis a symptom of 5-HT dysfunction??? Central Hyperacusis with phonophobia in multiple sclerosis.. Neurological conditions with Hyperacusis... Brainstem Lesion and Demyelinating Lesions in the Ipsilateral Pons and the Central Auditory Pathway... http://www.hyperacusis.org/wst_page5.html
|
|
|
|
|
Joined: May 2008
Posts: 4,707
Master Elite Member
|
Amalgam fillings and hearing loss
FORT COLLINS, COLORADO. The leaching of toxic mercury from amalgam fillings has been implicated in hearing loss. Mercury toxicity has also been linked to multiple sclerosis (MS). It is believed that the toxic effects of mercury cause damage to the blood brain barrier, demyelination (damage to the nerves' myelin sheaths) and slowing of the nerve conduction velocity. Now researchers at the Rocky Mountain Research Institute provide convincing proof that dental amalgam fillings may be responsible for the hearing loss often experienced by multiple sclerosis patients. Their experiment involved seven women aged 32-46 years who had been diagnosed with MS. The women underwent a standard hearing test in a sound booth and then had all their amalgam fillings replaced with composites. Six to eight months later they were again given the hearing test. Six of the seven patients had significantly improved hearing in the right ear and five of the seven showed improvement in the left ear. Overall, hearing improved an average of eight decibels. The researchers conclude that amalgam fillings may be a significant factor in hearing loss experienced by MS patients and could be a factor in hearing loss in other people as well.
Siblerud, Robert L. and Kienholz, Eldon. Evidence that mercury from dental amalgam may cause hearing loss in multiple sclerosis patients. Journal of Orthomolecular Medicine, Vol. 12, No. 4, Fourth Quarter, 1997, pp. 240-44
|
|
|
|
|
Joined: May 2008
Posts: 4,707
Master Elite Member
|
Multiple sclerosis is considered to be an autoimmune demyelinating disease of the central nervous system. autoimmune, inflammatory yet... But there is evidence suggesting that autoantibodies could play a role. Is there a connection seen with here with other inflammatory neurological disorders. A relationship? A connection? http://brain.oxfordjournals.org/content/127/2/269.full It said that the antibodies from the ADEM serum that bound to HOG cells were also mostly IgM. http://adem.org/Acute Disseminated Encephalomyelitis
|
|
|
|
|
