Still trying to paint the picture your way CTD.

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A while ago, both the observation and the prediction were disputed at EVC. RAZD participated in the discussion, and one can certainly make arguments both ways - either that he did not or did indeed take notes.
I participated in the thread but not in the discussion. You had enough to deal with from trixie and PaulK, who, interestingly took you to task for your claims about recombination.

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Surely a better, if downplayed, explanation of recombination can be found at http://www.pubmedcentral.nih.gov/articlerender.fcgi?artid=1526701
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THERE are two sources of genetic diversity, mutation and recombination. Mutation, broadly defined here as novel heritable change in nucleotide state, introduces new variants while recombination reassorts the variants along a chromosome into novel combinations or haplotypes. Recombination can occur through both homologous crossover and homologous (intralocus) gene conversion, processes that occur as part of meiosis in diploid (or higher ploidy) organisms (Wiuf and Hein 2000). Under the Holliday junction model (Holliday 1964), homologous gene conversion is thought to occur when only a short tract of the alternate chromosome (usually a few hundred base pairs) is incorporated during meiotic exchange (e.g., Stahl 1994).
Consider this fine point CTD:

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... recombination reassorts the variants along a chromosome into novel combinations or haplotypes.


Recombination sorts existing DNA between two existing (parent) strands into two new (offspring) strands. Where it starts on each strand is highly variable, and so you can have one offspring strand longer and the other shorter than they would be by a 1 for 1 switch. You also have no guarantee that they start and stop on whole genes rather than some arbitrary point on each strand.

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Mutation, broadly defined here as novel heritable change in nucleotide state, introduces new variants ...
So when recombination, because the start and stop points are not fixed in stone, results in novel heritable change in nucleotide states at the points of crossover, introducing new variants in the process,what do you have?

Mutation?
Recombination?
Or is this a false dichotomy?

Interestingly I started a thread on EvC Forum, "changes or mutations" where I tried to find distinctions of DNA change from mutations, and got taken to task by the geneticists there, who basically said that all changes were considered mutation. Most helpful for my understanding was Wounded King in post 16:

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So the range of mutation extends from single point mutations, through gene duplications and right the way up to whole genome duplications. A distinction might be made between sub-chromosomal and whole chromosomal level mutations. In which case you would have two categories of mutation one of which , the chromosomal and above, would be almost exclusively based on errors in the process of chromosome segregation durin cell division. All of the sub-chromosomal mutations not attributable to an ouside factor such as a retrovirus, would then be the result of changes brought about by a combination of chemical alterations to nucleotides and subsequent mis-repair or mis-copying, or the result of mis repair after more severe damage such as double stranded breaks, or the result of factors such as non-homologous recombination.
Note the bolded = mutation attributed to the recombination process.

Because recombination can result in novel change to DNA as well as restructuring, you do not have a black and white, one or the other, process. In fact the likelyhood of it being cut on strict genes on both strands is rather low, so the probability is that most recombination adds some novel DNA mix in the mix.

Enjoy.