Before my first time collapse I was able to bear hunger all day long without any problems at all. Neither felt I anything after a huge munching. On the contrary nowadays my symptoms enhance when I am hungry and I feel awful even after meal. Though Chlorella and Algin make the after meal symptoms a bit more bearable. So I am not sure if the main problem is the mercury, that is spilled into the intestine with the bile and gets redistributed. Algin and Chlorella should prevent mercury from redistribution, but they seem to fulfil only a part of the task.

As far as I know liver has two outlets. The above mentioned bile that gets pressed into the intestinal system and another route immediately into the blood circulation in case reserved glucose is needed. But what if this glucose is mercury contaminated? This could be an explanation why my symptoms enhance while being hungry.

On the other hand pancreas may also be contaminated. Enzymes produced by pancreas are excreted with the bile together which also should be cached by Algin and Chlorella. Pancreas produced insulin however gets immediately into the blood soon after glucose appears in the blood. This could be an explanation for my after meal symptoms.

Could anybody reinforce or refute my theory?